Download PDF by A. Bulikova : Antiphospholipid Syndrome
By A. Bulikova
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Extra resources for Antiphospholipid Syndrome
Despite this, there remains no alternative for clinicians who simply need accurate guidance in providing valid diagnoses for their patients. As previously mentioned, the potential APS clinical manifestations are numerous and widespread. This can be illustrated by listing a myriad of clinical specialities that can be involved in a patient’s managementrheumatology, neurology, cardiology, nephrology, endocrinology, gastroenterology, dermatology, surgery, haematology, intensive care, and obstetrics.
In one of these studies, a large kindred in which nine individuals had aPL antibodies was described. , 1990). A study also have described a family, including identical twins and their mother, in which all members had SLE and presented with different manifestations of APS. , 1993). , 1999). Thirty out of 101 family members from 7 families met diagnostic criteria for the syndrome. Segregation studies rejected environment and autosomal recessive models, and the data were fitted best by a dominant or codominant model.
2006) Pathogenic anti-beta2-glycoprotein I antibodies recognize domain I of beta2-glycoprotein I only after a conformational change. Blood. 107(5), 1916-1924. Eswaran K & Rosen SW. (1985) Recurrent abortions, thromboses, and a circulating anticoagulant. Am J Obst Gynecol. 151(6), 751-752. Galli M, Comfurius P, Maassen C, Hemker HC, de Baets MH, van Breda-Vriesman PJ, Barbui T, Zwaal RF & Bevers EM. (1990) Anticardiolipin antibodies (ACA) 32 Antiphospholipid Syndrome directed not to cardiolipin but to a plasma protein cofactor.
Antiphospholipid Syndrome by A. Bulikova